The Nervous System: A Three-volume Work Commemorating the 25th Anniversary of the National Institute of Neurological and Communicative Disorders and Stroke, Volume 2Donald B. Tower |
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Page 11
... onset in the prime of productive life , its pathologic or immunologic onset probably occurs at a younger age - perhaps appreciably younger . There may be several relatively common abor- tive forms of this disease , such as unilateral ...
... onset in the prime of productive life , its pathologic or immunologic onset probably occurs at a younger age - perhaps appreciably younger . There may be several relatively common abor- tive forms of this disease , such as unilateral ...
Page 51
... onset of symptoms , and virtually all were de- tected at approximately 28 months . About 67 % of astrocytomas had positive scans at the onset of symptoms . A maximum detection rate of 87 % was reached after approximately 28 months of ...
... onset of symptoms , and virtually all were de- tected at approximately 28 months . About 67 % of astrocytomas had positive scans at the onset of symptoms . A maximum detection rate of 87 % was reached after approximately 28 months of ...
Page 506
... onset REM periods we may look at other signs and symptoms of this entity ( notably , sleep paralysis and hypnagogic halluci- NORMAL SLEEP ONSET nations ) and build up a description and patho- physiological interpretation of the syndrome ...
... onset REM periods we may look at other signs and symptoms of this entity ( notably , sleep paralysis and hypnagogic halluci- NORMAL SLEEP ONSET nations ) and build up a description and patho- physiological interpretation of the syndrome ...
Contents
Influence of the National Institute of Neurological and Communicative | 1 |
Clinical Electroencephalography | 25 |
Neuroradiology as a Discipline | 35 |
Copyright | |
38 other sections not shown
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abnormalities activity acute agents aneurysms animals antibody antigen artery associated atherosclerosis biochemical blood flow brain cause cells cerebral atherosclerosis cerebral death cerebrospinal fluid changes chronic Clin clinical cortex cortical Creutzfeldt-Jakob disease defect deficiency dementia demonstrated diagnosis disease disorders dopamine drugs edited effects electrodes encephalopathy enzyme epilepsy epileptogenic experimental factors fibers focal frontal function Gaucher's disease genetic hemisphere hormone human hydrocephalus hypoxia ic iv impairment increased infection injury intracranial ischemia kuru L-DOPA lesions levels lipid lobe mechanisms membrane meningitis ment mental metabolism monkeys motor multiple sclerosis muscle myelin nerve nervous system Neurol neurological neuropathy Neurosurg normal occur onset pathological patients peripheral phosphenes phytanic acid population pressure protein rats Raven Press recent reduced reported response result scrapie seizures serum sleep specific spinal cord stimulation stroke studies surgical symptoms syndrome techniques Teuber therapy tion tissue treatment tumor vascular viral virus visual