The Nervous System: A Three-volume Work Commemorating the 25th Anniversary of the National Institute of Neurological and Communicative Disorders and Stroke, Volume 2Donald B. Tower |
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Page 118
... brain 10-3 pooled brain explants 100 days in vitro ic iv IC brain 10-3 IC lymph node + spleen 20 % brain 10-4 ic iv ic iv brain 10 : 4 ic iv brain 10-5 kidney 103 spleen 103 lymph node kidney + spleen positive transmission route IC ...
... brain 10-3 pooled brain explants 100 days in vitro ic iv IC brain 10-3 IC lymph node + spleen 20 % brain 10-4 ic iv ic iv brain 10 : 4 ic iv brain 10-5 kidney 103 spleen 103 lymph node kidney + spleen positive transmission route IC ...
Page 166
... Brain damage in the rhesus monkey resulting from profound arterial hypotension . I. Its nature , distribution and general physiological correlates . Brain Res . , 13 : 68–100 . 3. Paulson , O. B. ( 1971 ) : Cerebral apoplexy ( stroke ) ...
... Brain damage in the rhesus monkey resulting from profound arterial hypotension . I. Its nature , distribution and general physiological correlates . Brain Res . , 13 : 68–100 . 3. Paulson , O. B. ( 1971 ) : Cerebral apoplexy ( stroke ) ...
Page 386
... brain undergoes a series of changes which must occur in the proper sequence for adult structure and function to be normal . If malnutrition occurs at critical times during development , this sequence may be disrupted . The time when the ...
... brain undergoes a series of changes which must occur in the proper sequence for adult structure and function to be normal . If malnutrition occurs at critical times during development , this sequence may be disrupted . The time when the ...
Contents
Influence of the National Institute of Neurological and Communicative | 1 |
Clinical Electroencephalography | 25 |
Neuroradiology as a Discipline | 35 |
Copyright | |
38 other sections not shown
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abnormalities activity acute agents aneurysms animals antibody antigen artery associated atherosclerosis biochemical blood flow brain cause cells cerebral atherosclerosis cerebral death cerebrospinal fluid changes chronic Clin clinical cortex cortical Creutzfeldt-Jakob disease defect deficiency dementia demonstrated diagnosis disease disorders dopamine drugs edited effects electrodes encephalopathy enzyme epilepsy epileptogenic experimental factors fibers focal frontal function Gaucher's disease genetic hemisphere hormone human hydrocephalus hypoxia ic iv impairment increased infection injury intracranial ischemia kuru L-DOPA lesions levels lipid lobe mechanisms membrane meningitis ment mental metabolism monkeys motor multiple sclerosis muscle myelin nerve nervous system Neurol neurological neuropathy Neurosurg normal occur onset pathological patients peripheral phosphenes phytanic acid population pressure protein rats Raven Press recent reduced reported response result scrapie seizures serum sleep specific spinal cord stimulation stroke studies surgical symptoms syndrome techniques Teuber therapy tion tissue treatment tumor vascular viral virus visual